Hyperparathyroidism
- General information
- Increased secretion of PTH that results in an altered state of calcium, phosphate, and bone metabolism
- Most commonly affects women between ages 35-65
- Primary hyperparathyroidism: caused by tumor or hyperplasia of parathyroid glands
- Secondary hyperparathyroidism: caused by compensatory oversecretion of PTH in response to hypocalcemia from chronic renal disease, rickets, malabsorption syndrome, osteomalacia
- Assessment findings
- Bone pain (especially at back), bone demineralization, pathologic fractures
- Renal colic, kidney stones, polyuria, polydipsia
- Anorexia, nausea, vomiting, gastric ulcers, constipation
- Muscle weakness, fatigue
- Irritability, personality changes, depression
- Cardiac arrhythmias, hypertension
- Diagnostic tests
- Serum calcium levels elevated
- Serum phosphate levels decreased
- Skeletal x-rays reveal bone demineralization
- Nursing interventions
- Administer IV infusions of normal saline solution and give diuretics as ordered; monitor I&O and observe for fluid overload and electrolyte imbalances.
- Assist client with self-care: provide careful handling, moving, and ambulation to prevent pathologic fractures.
- Monitor vital signs; report irregularities.
- Force fluids; provide acid-ash juices, e.g., cranberry juice.
- Strain urine for stones.
- Provide low-calcium, high-phosphorus diet.
- Provide care for the client undergoing parathyroidectomy (see Thyroidectomy).
- Provide client teaching and discharge planning concerning
- Need to engage in progressive ambulatory activities
- Increased intake of fluids
- Use of calcium preparations and importance of high-calcium diet following a parathyroidectomy
Monday, May 19, 2008
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Labels:
endocrine disorder
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This entry was posted on Monday, May 19, 2008
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endocrine disorder
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