Cerebral Palsy (CP)
- General information
- Neuromuscular disorder resulting from damage to or altered structure of the part of the brain responsible for controlling motor function
- Incidence: 1.5-5 in 1,000 live births
- May be caused by a variety of factors resulting in damage to the CNS; possible causes include
- Prenatally: genetic, altered neurologic development, or trauma or anoxia to mother (toxemia, rubella, accidents)
- Perinatally: during the birth process (drugs at delivery, precipitate delivery, fetal distress, breech deliveries with delay)
- Postnatally: kernicterus or head trauma (child falls out of crib or is hit by a car)
- Medical management
- Drug therapy
- Antianxiety agents
- Skeletal muscle relaxants
- Local nerve blocks
- Physical/occupational therapy
- Speech/audiology therapy
- Surgery: muscle- and tendon-releasing procedures
- Assessment findings: disease itself does not progress once established; progressive complications, however, cause changes in signs and symptoms
- Spasticity: exaggerated hyperactive reflexes (increased muscle tone, increase in stretch reflex, scissoring of legs, poorly coordinated body movements for voluntary activities)
- Occurs with pyramidal tract lesion
- Found in 40% of all CP
- Results in contractures
- Also affects ability to speak: altered quality and articulation
- Loud noise or sudden movement causes reaction with increased spasm
- No parachute reflex to protect self when falling
- Athetosis: constant involuntary, purposeless, slow, writhing motions
- Occurs with extrapyramidal tract (basal ganglia) lesion
- Found in 40% of all CP
- Athetosis disappears during sleep, therefore contractures do not develop
- Movements increase with increase in physical or emotional stress
- Also affects facial muscles
- Ataxia: disturbance in equilibrium; diminished righting reflex (lack of balance, poor coordination, dizziness, hypotonia)
- Occurs with extrapyramidal tract (cerebellar) lesion
- Found in 10% of all CP
- Muscles and reflexes are normal
- Tremor: repetitive rhythmic involuntary contractions of flexor and extensor muscles
- Occurs with extrapyramidal tract (basal ganglia) lesion
- Found in 5% of all CP
- Interferes with performance of precise movements
- Often a mild disability
- Rigidity: resistance to flexion and extension resulting from simultaneous contraction of both agonist and antagonist muscle groups
- Occurs with extrapyramidal tract (basal ganglia) lesion
- Found in 5% of all CP
- Diminished or absent reflexes
- Potential for severe contractures
- Associated problems
- Mental retardation: the majority of CP clients are of normal or higher than average intelligence, but are unable to demonstrate it on standardized tests; 18%-50% have some form of mental retardation
- Hearing loss in 13% of CP clients
- Defective speech in 75% of CP clients
- Dental anomalies (from muscle contractures)
- Orthopedic problems from contractures or inability to mobilize
- Visual disabilities in 28% due to poor muscle control
- Disturbances of body image, touch, perception
- Feelings of worthlessness
- Nursing interventions
- Obtain a careful pregnancy, birth, and childhood history.
- Observe the child's behavior in various situations.
- Assist with activities of daily living (ADL), help child to learn as many self-care activities as possible; CP clients cannot do any task unless they are consciously aware of each step in the task; careful teaching and demonstration is essential.
- Provide a safe environment (safety helmet, padded crib).
- Provide physical therapy to prevent contractures and assist in mobility (braces if necessary).
- Provide client teaching and discharge planning concerning
- Nature of disease: CP is a nonfatal, noncurable disorder
- Need for continued physical, occupational, and speech therapy
- Care of orthopedic devices
- Provision for child's return to school
- Availability of support groups/community agencies.
Tuesday, May 20, 2008
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nervous disorder
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This entry was posted on Tuesday, May 20, 2008
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nervous disorder
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