SHOCK

An abnormal physiologic state where an imbalance between the amount of circulating blood volume and the size of the vascular bed results in circulatory failure and oxygen and nutrient deprivation of tissues. See Table 4.8 for classification of shock.

Body's Response to Shock

  1. Hyperventilation leading to respiratory alkalosis
  2. Vasoconstriction: shunts blood to heart and brain
  3. Tachycardia
  4. Fluid shifts: intracellular to extracellular shift to maintain circulating blood volume
  5. Impaired metabolism: tissue anoxia leads to anaerobic metabolism causing lactic acid buildup, resulting in metabolic acidosis
  6. Impaired organ function
    1. Kidney: decreased perfusion can result in renal failure.
    2. Lung: shock lung (adult respiratory distress syndrome [ARDS])



Table 4.8 Classifications of Shock

Type

Characteristics

Causes

Hypovolemic

Decreased circulating blood volume

Blood loss
Plasma loss (e.g., burns)
Fluid loss (e.g., excessive vomiting or diarrhea)

Cardiogenic

Failure of the heart to pump properly

Myocardial infarction
Congestive heart failure
Cardiac arrhythmias
Pericardial tamponade
Tension pneumothorax

Septic

Factors favoring septic shock:
* development of antibiotic-resistant organisms
* invasive procedures such as urinary tract of blood; results most frequently from gram-negative instrumentation
* immunosuppression and old age
* trauma: presence of blood in peritoneal cavity greatly increases likelihood of peritonitis

Release of bacterial toxins that act directly on the blood vessels producing massive vasodilation and pooling septicemia.

Neurogenic

Failure of arteriolar resistance, leading to massive vasodilation and pooling of blood

Interruption of sympathetic impulses from:
* exposure to unpleasant circumstances
* extreme pain
* spinal cord injury
* high spinal anesthesia
* vasomotor depression
* head injury

Anaphylactic

Massive vasodilation resulting from allergic reaction causing release of histamine and related substances

Allergic reaction to:
* insect venom
* medications
* dyes used in radiologic studies




Assessment Findings

  1. Skin
    1. Cool, pale, moist in hypovolemic and cardiogenic shock
    2. Warm, dry, pink in septic and neurogenic shock
  2. Pulse
    1. Tachycardia, due to increased sympathetic stimulation
    2. Weak and thready
  3. Blood pressure
    1. Early stages: may be normal due to compensatory mechanisms
    2. Later stages: systolic and diastolic blood pressure drops.
  4. Respirations: rapid and shallow, due to tissue anoxia and excessive amounts of CO2 (from metabolic acidosis)
  5. Level of consciousness: restlessness and apprehension, progressing to coma
  6. Urinary output: decreases due to impaired renal perfusion
  7. Temperature: decreases in severe shock (except septic shock).


Nursing Interventions

  1. Maintain patent airway and adequate ventilation.
    1. Establish and maintain airway.
    2. Administer oxygen as ordered.
    3. Monitor respiratory status, blood gases.
    4. Start resuscitative procedures as necessary.
  2. Promote restoration of blood volume; administer fluid and blood replacement as ordered
    1. Crystalloid solutions: Ringer's lactate, normal saline
    2. Colloid solutions: albumin, plasmanate, dextran
    3. Blood products: whole blood, packed red blood cells, fresh frozen plasma
  3. Administer drugs as ordered (see Table 4.9).
  4. Minimize factors contributing to shock.
    1. Elevate lower extremities to 45° to promote venous return to heart, thereby improving cardiac output.
    2. Avoid Trendelenburg's position: increases respiratory impairment.
    3. Promote rest by using energy-conservation measures and maintaining as quiet an environment as possible.
    4. Relieve pain by cautious use of narcotics.
      1. Since narcotics interfere with vasoconstriction, give only if absolutely necessary, IV and in small doses.
      2. If given IM or subcutaneously, vasoconstriction may cause incomplete absorption; when circulation improves, client may get overdose.
    5. Keep client warm.
  5. Maintain continuous assessment of the client.
    1. Check vital signs frequently.
    2. Monitor urine output: report urine output of less than 30 ml/hour.
    3. Observe color and temperature of skin.
    4. Monitor CVP.
    5. Monitor ECG.
    6. Check lab studies: CBC, electrolytes, BUN, creatinine, blood gases.
    7. Monitor other parameters such as arterial blood pressures, cardiac output, pulmonary artery pressures, pulmonary artery wedge pressures.
  6. Provide psychologic support: reassure client to relieve apprehension, and keep family advised.

Table 4.9 Drugs Used to Treat Shock


Generic (Trade) Name

Action

Dopamine (Intropin)

Low dosage: Dilates renal, mesenteric, and splanchnic vessels, which in turn increases perfusion of kidneys and urine output.
High dosage: Increases cardiac contractility; causes vasoconstriction (often given with nitroprusside [Nipride]).

Dobutamine (Dobutrex)

Increases myocardial contractility; vasodilator

Isoproterenol (Isuprel)

Increases myocardial contractility; decreases peripheral resistance by dilating peripheral vascular bed; usefulness is limited by the tachycardia it produces

Norepinephrine (Levophed)

Improves cardiac contractility and cardiac output; potent vasoconstrictor

Sodium nitroprusside (Nipride)

Vasodilator; decreases peripheral resistance and workload of heart, thereby increasing cardiac output; used in cardiogenic shock and hypertensive emergencies.

Digitalis preparations

Improve cardiac performance.

Corticosteroids

Used especially in septic shock; help to protect cell membranes and decrease the inflammatory response to stress.

Antibiotics

Used in treating infectious processes related to septic shock.

NOTE: Vasopressors such as Levophed can cause almost complete occlusion of arterioles, causing a decrease of blood flow to larger tissue areas. Therefore, if blood pressure is adequate, a vasodilator such as Nipride could probably be given as well, to modify the vasoconstrictor effects.

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